{"id":295,"date":"2013-10-29T23:09:04","date_gmt":"2013-10-29T23:09:04","guid":{"rendered":"http:\/\/www.mayohomeopathy.ie\/?p=295"},"modified":"2016-09-15T23:34:43","modified_gmt":"2016-09-15T22:34:43","slug":"sugar-harmful-not-saturated-fats","status":"publish","type":"post","link":"http:\/\/www.mayohomeopathy.ie\/index.php\/sugar-harmful-not-saturated-fats\/","title":{"rendered":"Sugar is harmful, not saturated fats says cardiologist"},"content":{"rendered":"
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sugar rationing, 1942-45<\/p><\/div>\n

Sugar Worse Than Fat<\/span><\/strong><\/h5>\n

Dr Aseem Malhotra, has caused a stir by raising concerns (he isn’t the first –\u00a0see my post on saturated fats and the Food Combining<\/em> book<\/a><\/span>)\u00a0over the “myth” that saturated fats do not cause heart disease, rather carbohydrates do by creating metabolic syndrome<\/em>. The result of this fallacy is that millions are unnecessarily prescribed statins (see my other posts on statins<\/a><\/span>). Being as powerful as the pharmaceutical industry, the sugar industry has enjoyed the benefits of the low fat era. In reducing fat from foods flavour is removed as fat gives flavour, so this deficiency is compensated for by adding sugar and other sweeteners. What happens to this sugar if not burned off? It’s stored as… fat! That’s apart from the damage done to arteries.\u00a0<\/span> Here is an earlier interview with\u00a0Dr Malhotra\u00a0on BBC;<\/span> http:\/\/youtu.be\/bgegpakWYVE<\/a>\u00a0<\/span><\/p>\n

“Experts” and “Lack of Science”<\/span><\/strong>
\nDr LeFanu<\/a> goes for the jugular as ever with a criticism of the lack of science behind the introduction of new health policies;<\/span><\/span><\/p>\n

The ‘expert committees’ that condemned butter and led eventually to the\u00a0over-prescription of statins were lacking the ‘habit of truth’… but the contrary holds where the surest way of promoting false\u00a0theories is by dressing them up in the garb of science. The favoured device, as many will know, is the \u201cexpert committee\u201d, where,\u00a0under the guise of dispassionately reviewing the relevant evidence, all\u00a0contrary findings and observations are omitted. Thus those with longer\u00a0memories may recall how 20 or so years ago, expert committees universally\u00a0concurred that the meat, milk and dairy foods produced by our hard-working\u00a0farmers were a potent cause of many forms of cancer and circulatory\u00a0disorders \u2013 and that switching to a low-fat diet would save \u201ctens of\u00a0\u00a0 thousands of lives\u201d.<\/span><\/p>\n

The critical tests of such extravagant claims \u2013 such as the lack of any\u00a0parallel between the incidence of these illnesses over time and the patterns\u00a0\u00a0 of food consumption \u2013 were ignored.<\/span><\/div>\n
More recently, this deceitful portrayal of milk, butter, eggs and so forth as\u00a0being uniquely harmful has, as the cardiologist Dr Aseem Malhotra points out\u00a0in the British Medical Journal, metamorphosed into recommendations by\u00a0similar groups of experts for the mass prescription of cholesterol-lowering\u00a0statins. In science, as elsewhere, \u201cthe habit of truth\u201d is not fostered by a\u00a0show of hands around a committee table.<\/span><\/div>\n<\/blockquote>\n
Scientific\u00a0Position Increased Cardiovascualr Risks<\/strong>
\nDr Malhotra’s\u00a0article,\u00a0Saturated Fat Is Not the Major Issue<\/a><\/span>,\u00a0explaining his position is published in the British Medical Journal which you can read below.<\/span><\/div>\n
\n

Scientists universally accept that trans fats\u2014found in many fast foods, bakery products, and margarines\u2014increase the risk of cardiovascular disease through inflammatory processes. But \u201csaturated fat\u201d is another story. The mantra that saturated fat must be removed to reduce the risk of cardiovascular disease has dominated dietary advice and guidelines for almost four decades.<\/span><\/p>\n

Yet scientific evidence shows that this advice has, paradoxically, increased our cardiovascular risks. Furthermore, the government\u2019s obsession with levels of total cholesterol, which has led to the overmedication of millions of people with statins, has diverted our attention from the more egregious risk factor of atherogenic dyslipidaemia.<\/span><\/p>\n

Saturated fat has been demonised ever since Ancel Keys\u2019s landmark \u201cseven countries\u201d study in 1970. This concluded that a correlation existed between the incidence of coronary heart disease and total cholesterol concentrations, which then correlated with the proportion of energy provided by saturated fat. But correlation is not causation. Nevertheless, we were advised to cut fat intake to 30% of total energy and saturated fat to 10%.\u201d The aspect of dietary saturated fat that is believed to have the greatest influence on cardiovascular risk is elevated concentrations of low density lipoprotein (LDL) cholesterol. Yet the reduction in LDL cholesterol from reducing saturated fat intake seems to be specific to large, buoyant (type A) LDL particles, when in fact it is the small, dense (type B) particles (responsive to carbohydrate intake) that are implicated in cardiovascular disease.<\/span><\/p>\n

Indeed, recent prospective cohort studies have not supported any significant association between saturated fat intake and cardiovascular risk. Instead, saturated fat has been found to be protective. The source of the saturated fat may be important. Dairy foods are exemplary providers of vitamins A and D. As well as a link between vitamin D deficiency and a significantly increased risk of cardiovascular mortality, calcium and phosphorus found commonly in dairy foods may have antihypertensive effects that may contribute to inverse associations with cardiovascular risk. One study showed that higher concentrations of plasma trans<\/em>-palmitoleic acid, a fatty acid mainly found in dairy foods, was associated with higher concentrations of high density lipoprotein, lower concentrations of triglycerides and C reactive protein, reduced insulin resistance, and a lower incidence of diabetes in adults. Red meat is another major source of saturated fat. Consumption of processed meats, but not red meat, has been associated with coronary heart disease and diabetes mellitus, which may be explained by nitrates and sodium as preservatives.<\/span><\/p>\n

The notoriety of fat is based on its higher energy content per gram in comparison with protein and carbohydrate. However, work by the biochemist Richard Feinman and nuclear physicist Eugene Fine on thermodynamics and the metabolic advantage of different diet compositions showed that the body did not metabolise different macronutrients in the same way. Kekwick and Pawan carried out one of the earliest obesity experiments, published in the Lancet<\/em> in 1956. They compared groups consuming diets of 90% fat, 90% protein, and 90% carbohydrate and showed that the greatest weight loss was in the fat consuming group. The authors concluded that the \u201ccomposition of the diet appeared to outweigh in importance the intake of calories.\u201d<\/span><\/p>\n

The \u201ccalorie is not a calorie\u201d theory has been further substantiated by a recent JAMA<\/em> study showing that a \u201clow fat\u201d diet resulted in the greatest decrease in energy expenditure, an unhealthy lipid pattern, and increased insulin resistance in comparison with a low carbohydrate and low glycaemic index diet. In the past 30 years in the United States the proportion of energy from consumed fat has fallen from 40% to 30% (although absolute fat consumption has remained the same), yet obesity has rocketed.<\/span><\/p>\n

One reason: when you take the fat out, the food tastes worse. The food industry compensated by replacing saturated fat with added sugar. The scientific evidence is mounting that sugar is a possible independent risk factor for the metabolic syndrome (the cluster of hypertension, dysglycaemia, raised triglycerides, low HDL cholesterol, and increased waist circumference).<\/span><\/p>\n

In previous generations cardiovascular disease existed largely in isolation. Now two thirds of people admitted to hospital with a diagnosis of acute myocardial infarction really have metabolic syndrome\u2014but 75% of these patients have completely normal total cholesterol concentrations. Maybe this is because total cholesterol isn\u2019t really the problem?<\/span><\/p>\n

The Framingham heart study sanctified total cholesterol as a risk factor for coronary artery disease, making statins the second most prescribed drug in the US and driving a multibillion dollar global industry. In the United Kingdom eight million people take statins regularly, up from five million 10 years ago. With 60 million statin prescriptions a year, it is difficult to demonstrate any additional effect of statins on reduced cardiovascular mortality over the effects of the decline in smoking prevalence and primary angioplasty.<\/span><\/p>\n

Despite the common belief that high cholesterol is a significant risk factor for coronary artery disease, several independent population studies in healthy adults have shown that low total cholesterol is associated with cardiovascular and non-cardiac mortality, indicating that high total cholesterol is not a risk factor in a healthy population.<\/span><\/p>\n

A recent \u201creal world\u201d study of 150\u2009000 patients who were taking statins showed \u201cunacceptable\u201d side effects\u2014including myalgia, gastrointestinal upset, sleep and memory disturbance, and erectile dysfunction\u2014in 20% of participants, resulting in discontinuation of the drug. This is massively at odds with the major statin trials that report significant side effects of myopathy or muscle pain in only one in 10\u2009000.<\/span><\/p>\n

A meta-analysis of predominantly industry sponsored data reported that in a low risk group of people aged 60-70 years taking statins the number needed to treat (NNT) to prevent one cardiovascular event in one year was 345. The strongest evidence base for statins is in secondary prevention, where all patients after a myocardial infarction are prescribed maximum dose treatment irrespective of total cholesterol, because of statins\u2019 anti-inflammatory or pleiotropic (coronary plaque stabilising) effects. In this group the NNT is 83 for mortality over five years. This doesn\u2019t mean that each patient benefits a little but rather that 82 will receive no prognostic benefit. The fact that no other cholesterol lowering drug has shown a benefit in terms of mortality supports the hypothesis that the benefits of statins are independent of their effects on cholesterol.<\/span><\/p>\n

Adopting a Mediterranean diet after a heart attack is almost three times as powerful in reducing mortality as taking a statin. The recently published PREDIMED randomised controlled trial was stopped early after it showed that in high risk people the Mediterranean diet achieved a 30% improvement over a \u201clow fat\u201d diet in terms of cardiovascular events.<\/span><\/p>\n

Pharmacotherapy can assuage the symptoms but can\u2019t alter the pathophysiology. Doctors need to embrace prevention as well as treatment. The greatest improvements in morbidity and mortality have been due not to personal responsibility but rather to public health. It is time to bust the myth of the role of saturated fat in heart disease and wind back the harms of dietary advice that has contributed to obesity.<\/span><\/p>\n<\/blockquote>\n

Updates<\/span><\/strong>
\nJanuary 2016: Dr Malhotra suggests an apple a day is as effective as statins.<\/span> See my post here<\/a>\u00a0and this commentary in the<\/span> Daily Telegraph<\/a><\/span><\/p>\n

We need a healthy dose of lifestyle medicine and less drugs<\/a><\/span>, by Dr Aseem Malhotra who criticises the current medical approach to diabetes and cholesterol treatment.<\/span><\/p>\n

September 2016. It’s just come to light the sugar industry paid Harvard “researchers” to shift the blame to the fat industry. Read about it here<\/a><\/span>.<\/span><\/p>\n

Photo credits: WikiCommons<\/a><\/span><\/span><\/p>\n","protected":false},"excerpt":{"rendered":"

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